Listar por autor "Soria Valles, Clara"

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Analysis of the Disintegrin-metalloproteinases Family Reveals ADAM29 and ADAM7 Are Often Mutated in Melanoma

Wei, Xiaomu; Moncada Pazos, Ángela; Cal Miguel, Santiago Jesús; Soria Valles, Clara; Gartner, Jared; Rudloff, Udo; Lin, Jimmy C.; Rosenberg, Steven A.; López Otín, Carlos; Samuels, Yardena; NISC Comparative Sequencing (Wiley-Liss, 2011)
The anti-metastatic activity of collagenase-2 in breast cancer cells is mediated by a signaling pathway involving decorin and miR-21

Soria Valles, Clara; Gutiérrez Fernández, Ana Yolanda; Guiu, M.; Mari, B.; Fueyo Casado, Alejandro; Gomis, R. R.; López Otín, Carlos (2014)
Antioxidant responses to variations of oxygen by the Harderian gland of different species of the superspecies Spalax ehrenbergi

Soria Valles, Clara; Caballero García, Beatriz; Vega Naredo, Ignacio; Sierra Sánchez, Verónica; Huidobro Fernández, Covadonga; Gonzalo Calvo, David de; Tolivia Cadrecha, Delio Rubén; Rodríguez Colunga, María Josefa; Joel, Alma; Coto Montes, Ana María; Avivi, Aaron (2010)
Correction: The anti-metastatic activity of collagenase-2 in breast cancer cells is mediated by a signaling pathway involving decorin and miR-21 (Oncogene, (2014), 33, 23, (3054-3063), 10.1038/onc.2013.267)

Soria Valles, Clara; Gutiérrez Fernández, Ana Yolanda; Guiu, M.; Mari, B.; Fueyo Silva, Antonio Manuel; Gomis, R. R.; López Otín, Carlos (2019)
Effects of azacitidine on matrix metalloproteinase-9 in acute myeloid leukemia and myelodysplasia

Bernal del Castillo, Teresa; Moncada Pazos, Ángela; Soria Valles, Clara; Gutiérrez Fernández, Ana Yolanda (2013)
Erratum to: NF-¿B activation impairs somatic cell reprogramming in ageing (Nature Cell Biology, (2015), 17, 8, (1004-1013), 10.1038/ncb3207)

Soria Valles, Clara; García Osorio, Fernando; Gutiérrez Fernández, Ana Yolanda; De los Ángeles, A.; Bueno, Clara; Menéndez, P.; Martín Subero, J. I.; Daley, G. Q.; Pérez Freije, José María; López Otín, Carlos (2018)
Favorable effects of a prolonged treatment with melatonin on the level of oxidative damage and neurodegeneration in senescence-accelerated mice

Caballero García, Beatriz; Vega Naredo, Ignacio; Sierra Sánchez, Verónica; Huidobro Fernández, Covadonga; Soria Valles, Clara; Gonzalo Calvo, David de; Tolivia Fernández, Delio Rubén; Gutiérrez Cuesta, Javier; Pallàs Lliberia, Mercè; Camins Espuny, Antoni; Rodríguez Colunga, María Josefa; Coto Montes, Ana María (2008)
Hallmarks of progeroid syndromes: Lessons from mice and reprogrammed cells

Carrero Muñiz, Dido; Soria Valles, Clara; López Otín, Carlos (2016)
Immune and inflammatory responses to DNA damage in cancer and aging

Soria Valles, Clara; López Soto, Alejandro; García Osorio, Fernando; López Otín, Carlos (2017)
IPSCs: On the Road to Reprogramming Aging

Soria Valles, Clara; López Otín, Carlos (2016)
Loss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acid

Gutiérrez Fernández, Ana Yolanda; Soria Valles, Clara; García Osorio, Fernando; Gutiérrez Abril, Jesús; Garabaya Fernández, Cecilia; Aguirre Quevedo, Alina; Fueyo Silva, Antonio Manuel; Fernández García, María de la Soledad; Suárez Puente, Xosé Antón; López Otín, Carlos (2015)
Loss of the proteostasis factor AIRAPL causes myeloid transformation by deregulating IGF-1 signaling

García Osorio, Fernando; Soria Valles, Clara; Santiago Fernández, Olaya; Pérez Freije, José María; López Otín, Carlos (2016)
Melatonin alters cell death processes in response to age-related oxidative stress in the brain of senescence-accelerated mice

Caballero García, Beatriz; Vega Naredo, Ignacio; Sierra Sánchez, Verónica; Huidobro Fernández, Covadonga; Soria Valles, Clara; Gonzalo Calvo, David de; Tolivia Fernández, Delio Rubén; Pallàs Lliberia, Mercè; Camins Espuny, Antoni; Rodríguez Colunga, María Josefa; Coto Montes, Ana María (Blackwell Publishing Ltd, 2009)
MMP-25 metalloprotease regulates innate immune response through NF-κB signaling

Soria Valles, Clara; Gutiérrez Fernández, Ana Yolanda; García Osorio, Fernando; Carrero Muñiz, Dido; Ferrando, Adolfo A.; Colado Varela, Enrique; Fernández García, María de la Soledad; Bonzon-Kulichenko, E.; Vázquez, Jesús; Fueyo Silva, Antonio Manuel; López Otín, Carlos (2016)
NF-?B activation impairs somatic cell reprogramming in ageing

Soria Valles, Clara; García Osorio, Fernando; Gutiérrez Fernández, Ana Yolanda; Ángeles, A. de los; Bueno, Clara; Menéndez, P.; Martín Subero, J. I.; Daley, G. Q.; Pérez Freije, José María; López Otín, Carlos (2015)
NF-κB signaling as a driver of ageing

García Osorio, Fernando; Soria Valles, Clara; Santiago Fernández, Olaya; Pérez Freije, José María; López Otín, Carlos (2016)
Novel LMNA mutations cause an aggressive atypical neonatal progeria without progerin accumulation

Soria Valles, Clara; Carrero Muñiz, Dido; Gabau, E.; Velasco Cotarelo, María Gloria; Quesada Fernández, Víctor; Bárcena Fernández, Clea; Moens, M.; Fieggen, K.; Möhrcken, S.; Owens, M.; Álvarez Puente, Diana; Asensio, O.; Loeys, B.; Pérez, Ana; Benoit, V.; Wuyts, W.; Lévy, Nicolas; Hennekam, R. C.; Sandre-Giovannoli, Annachiara de; López Otín, Carlos (2016)
Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response

García Osorio, Fernando; Bárcena Fernández, Clea; Soria Valles, Clara; Ramsay, Andrew John; Carlos Villafranca, Félix Antonio de; Cobo Plana, Juan Manuel; Fueyo Silva, Antonio Manuel; Pérez Freije, José María; López Otín, Carlos (American Association of Immunologists, 2012)
Reprogramming aging through DOT1L inhibition

Soria Valles, Clara; García Osorio, Fernando; López Otín, Carlos (2015)
Sexual autophagic differences in the androgen-dependent flank organ of Syrian hamster

Coto Montes, Ana María; Tomás Zapico, Cristina; Martínez Fraga, Jorge; Vega Naredo, Ignacio; Sierra Sánchez, Verónica; Caballero García, Beatriz; Huidobro Fernández, Covadonga; Soria Valles, Clara; Tolivia Cadrecha, Delio Rubén; Rodríguez Colunga, María Josefa (2009)

Listar por autor "Soria Valles, Clara"

Analysis of the Disintegrin-metalloproteinases Family Reveals ADAM29 and ADAM7 Are Often Mutated in Melanoma ﻿

The anti-metastatic activity of collagenase-2 in breast cancer cells is mediated by a signaling pathway involving decorin and miR-21 ﻿

Antioxidant responses to variations of oxygen by the Harderian gland of different species of the superspecies Spalax ehrenbergi ﻿

Correction: The anti-metastatic activity of collagenase-2 in breast cancer cells is mediated by a signaling pathway involving decorin and miR-21 (Oncogene, (2014), 33, 23, (3054-3063), 10.1038/onc.2013.267) ﻿

Effects of azacitidine on matrix metalloproteinase-9 in acute myeloid leukemia and myelodysplasia ﻿

Erratum to: NF-¿B activation impairs somatic cell reprogramming in ageing (Nature Cell Biology, (2015), 17, 8, (1004-1013), 10.1038/ncb3207) ﻿

Favorable effects of a prolonged treatment with melatonin on the level of oxidative damage and neurodegeneration in senescence-accelerated mice ﻿

Hallmarks of progeroid syndromes: Lessons from mice and reprogrammed cells ﻿

Immune and inflammatory responses to DNA damage in cancer and aging ﻿

IPSCs: On the Road to Reprogramming Aging ﻿

Loss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acid ﻿

Loss of the proteostasis factor AIRAPL causes myeloid transformation by deregulating IGF-1 signaling ﻿

Melatonin alters cell death processes in response to age-related oxidative stress in the brain of senescence-accelerated mice ﻿

MMP-25 metalloprotease regulates innate immune response through NF-κB signaling ﻿

NF-?B activation impairs somatic cell reprogramming in ageing ﻿

NF-κB signaling as a driver of ageing ﻿

Novel LMNA mutations cause an aggressive atypical neonatal progeria without progerin accumulation ﻿

Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response ﻿

Reprogramming aging through DOT1L inhibition ﻿

Sexual autophagic differences in the androgen-dependent flank organ of Syrian hamster ﻿

Analysis of the Disintegrin-metalloproteinases Family Reveals ADAM29 and ADAM7 Are Often Mutated in Melanoma

The anti-metastatic activity of collagenase-2 in breast cancer cells is mediated by a signaling pathway involving decorin and miR-21

Antioxidant responses to variations of oxygen by the Harderian gland of different species of the superspecies Spalax ehrenbergi

Correction: The anti-metastatic activity of collagenase-2 in breast cancer cells is mediated by a signaling pathway involving decorin and miR-21 (Oncogene, (2014), 33, 23, (3054-3063), 10.1038/onc.2013.267)

Effects of azacitidine on matrix metalloproteinase-9 in acute myeloid leukemia and myelodysplasia

Erratum to: NF-¿B activation impairs somatic cell reprogramming in ageing (Nature Cell Biology, (2015), 17, 8, (1004-1013), 10.1038/ncb3207)

Favorable effects of a prolonged treatment with melatonin on the level of oxidative damage and neurodegeneration in senescence-accelerated mice

Hallmarks of progeroid syndromes: Lessons from mice and reprogrammed cells

Immune and inflammatory responses to DNA damage in cancer and aging

IPSCs: On the Road to Reprogramming Aging

Loss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acid

Loss of the proteostasis factor AIRAPL causes myeloid transformation by deregulating IGF-1 signaling

Melatonin alters cell death processes in response to age-related oxidative stress in the brain of senescence-accelerated mice

MMP-25 metalloprotease regulates innate immune response through NF-κB signaling

NF-?B activation impairs somatic cell reprogramming in ageing

NF-κB signaling as a driver of ageing

Novel LMNA mutations cause an aggressive atypical neonatal progeria without progerin accumulation

Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response

Reprogramming aging through DOT1L inhibition

Sexual autophagic differences in the androgen-dependent flank organ of Syrian hamster