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Nuclear lamina defects cause ATM-dependent NF-κB activation and link accelerated aging to a systemic inflammatory response

Author:
García Osorio, Fernandountranslated; Bárcena Fernández, Cleauntranslated; Soria Valles, Clarauntranslated; Ramsay, Andrew Johnuntranslated; Carlos Villafranca, Félix Antonio deuntranslated; Cobo Plana, Juan Manueluntranslated; Fueyo Silva, Antonio Manueluntranslated; Pérez Freije, José Maríauntranslated; López Otín, Carlosuntranslated
Publication date:
2012
Editorial:

American Association of Immunologists
Publisher version:
http://dx.doi.org/10.1101/gad.197954.112
Citación:
Genes and Development, 26(20), p. 2311-2324 (2012); doi:10.1101/gad.197954.112
Descripción física:
p. 2311-2324
URI:
http://hdl.handle.net/10651/6477
ISSN:
0890-9369
Identificador local:

20121269
DOI:
10.1101/gad.197954.112
Patrocinado por:

This work was supported by grants from Ministerio de Ciencia e Innovación-Spain and European Union (FP7 MicroEnviMet). The Instituto Universitario de Oncología is supported by Obra Social Cajastur and Acción Transversal del Cáncer-RTICC. C.L.-O. is an Investigator of the Botin Foundation
Id. Proyecto:

info:eu-repo/grantAgreement/EC/FP7/201279
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© 2012 by Cold Spring Harbor Laboratory Press
This item is protected with a Creative Commons License