Listar por autor "Aguirre Quevedo, Alina"

Anti-inflammatory effects of clarithromycin in ventilator-induced lung injury

Amado Rodríguez, Laura

; González López, Adrián

; López Alonso, Inés

; Aguirre Quevedo, Alina

; Astudillo González, María Aurora

; Batalla Solís, Estefanía

; Blázquez Prieto, Jorge

; García Prieto, Emilio

; Muñiz Albaiceta, Guillermo

(BioMedCentral, 2013)

ATG4B/autophagin-1 regulates intestinal homeostasis and protects mice from experimental colitis

Cabrera Benítez, María Sandra

; Fernández Fernández, Álvaro

; Mariño García, Guillermo

; Aguirre Quevedo, Alina

; Fernández Suárez, María

; Español Fernández, Yaiza

; Vega Álvarez, José Antonio

; Laurà, Rosaria; Fueyo Silva, Antonio Manuel

; Fernández-García, Soledad; Pérez Freije, José María

; Kroemer, Guido; López Otín, Carlos

(2013)

Defective autophagy impairs ATF3 activity and worsens lung injury during endotoxemia

Aguirre Quevedo, Alina

; López Alonso, Inés

; González López, Adrián

; Amado Rodríguez, Laura

; Batalla Solís, Estefanía

; Astudillo González, María Aurora

; Blázquez Prieto, Jorge

; Fernández Fernández, Álvaro

; Galván Hernández, José Alberto

; Dos Santos, Claudia C.; Muñiz Albaiceta, Guillermo

(2014)

Dual dose-related effects evoked by CCL4 on thermal nociception after gene delivery or exogenous administration in mice

Aguirre Quevedo, Alina

; González Rodríguez, Sara

; García Domínguez, Mario

; Lastra Queipo, Ana María

; Gutiérrez Fernández, Ana Yolanda

; Hidalgo Balsera, Agustín

; Menéndez Antolín, Luis

; Baamonde Arbaiza, Ana Isabel

(2020)

Impairment of autophagy decreases ventilator-induced lung injury by blockade of the NF-kappa B pathway

López Alonso, Inés

; Aguirre Quevedo, Alina

; González López, Adrián

; Fernández Fernández, Álvaro

; Amado Rodríguez, Laura

; Astudillo González, María Aurora

; Batalla Solís, Estefanía

; Muñiz Albaiceta, Guillermo

(2013)

Implicación de diferentes sistemas proteolíticos en modelos de lesión pulmonar aguda

Aguirre Quevedo, Alina

(2014-07-03)

Loss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acid

Gutiérrez Fernández, Ana Yolanda

; Soria Valles, Clara

; García Osorio, Fernando

; Gutiérrez Abril, Jesús

; Garabaya Fernández, Cecilia

; Aguirre Quevedo, Alina

; Fueyo Silva, Antonio Manuel

; Fernández García, María de la Soledad

; Suárez Puente, Xosé Antón

; López Otín, Carlos

(2015)

Matrix metalloproteinase-14 triggers an anti-inflammatory proteolytic cascade in endotoxemia

Aguirre Quevedo, Alina

; Blázquez Prieto, Jorge

; Amado Rodríguez, Laura

; López Alonso, Inés

; Batalla Solís, Estefanía

; González López, Adrián

; Sánchez Pérez, Moisés; Mayoral García, Carlos; Gutiérrez Fernández, Ana Yolanda

; Muñiz Albaiceta, Guillermo

(Springer Verlag, 2017)

Mechanical ventilation triggers hippocampal apoptosis by vagal and dopaminergic pathways

González López, Adrián

; López Alonso, Inés

; Aguirre Quevedo, Alina

; Amado Rodríguez, Laura

; Batalla Solís, Estefanía

; Astudillo González, María Aurora

; Tomás Zapico, Cristina

; Fueyo, Antonio; Dos Santos, Claudia C.; Talbot, Konrad; Muñiz Albaiceta, Guillermo

(2013)

MMP-8 deficiency increases TLR/RAGE ligands S100A8 and S100A9 and exacerbates lung inflammation during endotoxemia

González López, Adrián

; Aguirre Quevedo, Alina

; López Alonso, Inés

; Amado Rodríguez, Laura

; Astudillo González, María Aurora

; Fernández García, María de la Soledad

; Fernández Suárez, María

; Batalla Solís, Estefanía

; Colado Varela, Enrique; Muñiz Albaiceta, Guillermo

(2012)

The Systemic Administration of the Chemokine CCL1 Evokes Thermal Analgesia in Mice Through the Activation of the Endocannabinoid System

García Domínguez, Mario

; Aguirre Quevedo, Alina

; Lastra Queipo, Ana María

; Hidalgo Balsera, Agustín

; Baamonde Arbaiza, Ana Isabel

; Menéndez Antolín, Luis

(2019)

Listar por autor "Aguirre Quevedo, Alina"

Anti-inflammatory effects of clarithromycin in ventilator-induced lung injury ﻿

ATG4B/autophagin-1 regulates intestinal homeostasis and protects mice from experimental colitis ﻿

Defective autophagy impairs ATF3 activity and worsens lung injury during endotoxemia ﻿

Dual dose-related effects evoked by CCL4 on thermal nociception after gene delivery or exogenous administration in mice ﻿

Impairment of autophagy decreases ventilator-induced lung injury by blockade of the NF-kappa B pathway ﻿

Implicación de diferentes sistemas proteolíticos en modelos de lesión pulmonar aguda ﻿

Loss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acid ﻿

Matrix metalloproteinase-14 triggers an anti-inflammatory proteolytic cascade in endotoxemia ﻿

Mechanical ventilation triggers hippocampal apoptosis by vagal and dopaminergic pathways ﻿

MMP-8 deficiency increases TLR/RAGE ligands S100A8 and S100A9 and exacerbates lung inflammation during endotoxemia ﻿

The Systemic Administration of the Chemokine CCL1 Evokes Thermal Analgesia in Mice Through the Activation of the Endocannabinoid System ﻿

Anti-inflammatory effects of clarithromycin in ventilator-induced lung injury

ATG4B/autophagin-1 regulates intestinal homeostasis and protects mice from experimental colitis

Defective autophagy impairs ATF3 activity and worsens lung injury during endotoxemia

Dual dose-related effects evoked by CCL4 on thermal nociception after gene delivery or exogenous administration in mice

Impairment of autophagy decreases ventilator-induced lung injury by blockade of the NF-kappa B pathway

Implicación de diferentes sistemas proteolíticos en modelos de lesión pulmonar aguda

Loss of MT1-MMP causes cell senescence and nuclear defects which can be reversed by retinoic acid

Matrix metalloproteinase-14 triggers an anti-inflammatory proteolytic cascade in endotoxemia

Mechanical ventilation triggers hippocampal apoptosis by vagal and dopaminergic pathways

MMP-8 deficiency increases TLR/RAGE ligands S100A8 and S100A9 and exacerbates lung inflammation during endotoxemia

The Systemic Administration of the Chemokine CCL1 Evokes Thermal Analgesia in Mice Through the Activation of the Endocannabinoid System