Listar por autor "Carrero Muñiz, Dido"

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Comparative genomics of mortal and immortal cnidarians unveils novel keys behind rejuvenation

Pascual Torner, María; Carrero Muñiz, Dido; Pérez Silva, J. G.; Álvarez Puente, Diana; Roiz del Valle, David; Bretones Sánchez, Gabriel; Rodríguez Martínez, David; Maeso Miguel, Daniel; Mateo González, Elena; Español Fernández, Yaiza; Mariño García, Guillermo; Acuña Fernández, José Luis; Quesada Fernández, Víctor; López Otín, Carlos (2022)
Differential mechanisms of tolerance to extreme environmental conditions in tardigrades

Carrero Muñiz, Dido; González Pérez-Silva, José María; Quesada Fernández, Víctor; López Otín, Carlos (2019)
Genomic and functional analysis of longevity and stress resistance determinants in Metazoans

Carrero Muñiz, Dido (2022-03-18)
Giant tortoise genomes provide insights into longevity and age-related disease

Quesada Fernández, Víctor; Freitas Rodríguez, Sandra; Miller, J.; Santiago Fernández, Olaya; Campos Iglesias, Diana; Carrero Muñiz, Dido; Araujo Voces, Miguel; Mayoral García, Pablo; Rodríguez Arango, Javier; Tamargo Gómez, Isaac; Pascual Torner, María; López Otín, Carlos (2019)
Hallmarks of progeroid syndromes: Lessons from mice and reprogrammed cells

Carrero Muñiz, Dido; Soria Valles, Clara; López Otín, Carlos (2016)
MMP-25 metalloprotease regulates innate immune response through NF-κB signaling

Soria Valles, Clara; Gutiérrez Fernández, Ana Yolanda; García Osorio, Fernando; Carrero Muñiz, Dido; Ferrando, Adolfo A.; Colado Varela, Enrique; Fernández García, María de la Soledad; Bonzon-Kulichenko, E.; Vázquez, Jesús; Fueyo Silva, Antonio Manuel; López Otín, Carlos (2016)
Novel LMNA mutations cause an aggressive atypical neonatal progeria without progerin accumulation

Soria Valles, Clara; Carrero Muñiz, Dido; Gabau, E.; Velasco Cotarelo, María Gloria; Quesada Fernández, Víctor; Bárcena Fernández, Clea; Moens, M.; Fieggen, K.; Möhrcken, S.; Owens, M.; Álvarez Puente, Diana; Asensio, O.; Loeys, B.; Pérez, Ana; Benoit, V.; Wuyts, W.; Lévy, Nicolas; Hennekam, R. C.; Sandre-Giovannoli, Annachiara de; López Otín, Carlos (2016)
The relevance of induced pluripotent stem cells for the study of physiological and premature aging

Carrero Muñiz, Dido; López Otín, Carlos (2018)
Specific combinations of biallelic POLR3A variants cause Wiedemann-Rautenstrauch syndrome

Paolacci, Stefano; Li, Yun; Agolini, Emanuele; Carrero Muñiz, Dido; Velasco Cotarelo, María Gloria; López Otín, Carlos (2018)

Listar por autor "Carrero Muñiz, Dido"

Comparative genomics of mortal and immortal cnidarians unveils novel keys behind rejuvenation ﻿

Differential mechanisms of tolerance to extreme environmental conditions in tardigrades ﻿

Genomic and functional analysis of longevity and stress resistance determinants in Metazoans ﻿

Giant tortoise genomes provide insights into longevity and age-related disease ﻿

Hallmarks of progeroid syndromes: Lessons from mice and reprogrammed cells ﻿

MMP-25 metalloprotease regulates innate immune response through NF-κB signaling ﻿

Novel LMNA mutations cause an aggressive atypical neonatal progeria without progerin accumulation ﻿

The relevance of induced pluripotent stem cells for the study of physiological and premature aging ﻿

Specific combinations of biallelic POLR3A variants cause Wiedemann-Rautenstrauch syndrome ﻿

Comparative genomics of mortal and immortal cnidarians unveils novel keys behind rejuvenation

Differential mechanisms of tolerance to extreme environmental conditions in tardigrades

Genomic and functional analysis of longevity and stress resistance determinants in Metazoans

Giant tortoise genomes provide insights into longevity and age-related disease

Hallmarks of progeroid syndromes: Lessons from mice and reprogrammed cells

MMP-25 metalloprotease regulates innate immune response through NF-κB signaling

Novel LMNA mutations cause an aggressive atypical neonatal progeria without progerin accumulation

The relevance of induced pluripotent stem cells for the study of physiological and premature aging

Specific combinations of biallelic POLR3A variants cause Wiedemann-Rautenstrauch syndrome