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Prelamin A causes progeria through cell-extrinsic mechanisms and prevents cancer invasion

dc.contributor.authorRosa de Sáa, Jorge de la
dc.contributor.authorPérez Freije, José María 
dc.contributor.authorCabanillas Farpón, Rubén 
dc.contributor.authorGarcía Osorio, Fernando 
dc.contributor.authorFernández Fraga, Mario 
dc.contributor.authorFernández García, María de la Soledad 
dc.contributor.authorRad, Roland
dc.contributor.authorFanjul Hevia, Víctor
dc.contributor.authorPiñeiro Ugalde, Alejandro 
dc.contributor.authorLiang, Qi
dc.contributor.authorProsser, Haydn M.
dc.contributor.authorBradley, Allan
dc.contributor.authorCadiñanos Bañales, Juan
dc.contributor.authorLópez Otín, Carlos 
dc.date.accessioned2013-11-27T11:29:29Z
dc.date.available2013-11-27T11:29:29Z
dc.date.issued2013
dc.identifier.citationNature Communications, 4, p. 2268-2268 (2013); doi:10.1038/ncomms3268
dc.identifier.issn2041-1723
dc.identifier.urihttp://hdl.handle.net/10651/19812
dc.description.sponsorshipThis work was supported by grants from Ministerio de Economía y Competitividad-Spain (grant nos MICINN-12-SAF2011-23089 and PI12/01080), European Union (FP7 MicroEnviMet; grant no. UE-08-FP7-HEALTH-2007-A) and the Wellcome Trust (grant no. 079643), as well as by Fundación María Cristina Masaveu Peterson, Fundación Centro Médico de Asturias, Obra Social Cajastur and Fundación Ramón Areces (grant no. SV-12-Areces-1). IUOPA is supported by Obra Social Cajastur and RTICC (Instituto Carlos III, Madrid, Spain). C.L.-O. is an Investigator of the Botín Foundation. J.C. was a recipient of a FEBS Long-term fellowship during part of this work. J.d.l.R. is a recipient of a predoctoral fellowship from Fundación María Cristina Masaveu Peterson.
dc.format.extentp. 2268-2268
dc.language.isoeng
dc.relation.ispartofNature Communications
dc.rights© 2013 Macmillan Publishers Limited.
dc.rightsCC Reconocimiento - No comercial - Sin obras derivadas 3.0
dc.rights.urihttp://creativecommons.org/licenses/by/3.0/
dc.sourceWOK
dc.titlePrelamin A causes progeria through cell-extrinsic mechanisms and prevents cancer invasion
dc.typejournal article
dc.identifier.local20131024
dc.identifier.doi10.1038/ncomms3268
dc.relation.projectIDMICINN-12-SAF2011-23089
dc.relation.projectIDPI12/01080
dc.relation.projectIDSV-12-Areces-1
dc.relation.projectIDUE-08-FP7-HEALTH-2007-A
dc.relation.publisherversionhttp://dx.doi.org/10.1038/ncomms3268
dc.rights.accessRightsopen access


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