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Why have transgenic rodent models failed to successfully mimic Alzheimer’s disease. How can we develop effective drugs without them?

dc.contributor.authorFranco, R.
dc.contributor.authorMartínez Pinilla, Eva 
dc.contributor.authorNavarro, G.
dc.date.accessioned2019-08-21T07:34:05Z
dc.date.available2019-08-21T07:34:05Z
dc.date.issued2019
dc.identifier.citationExpert Opinion on Drug Discovery, 14(4), p. 327-330 (2019); doi:10.1080/17460441.2019.1581169
dc.identifier.issn1746-0441
dc.identifier.urihttp://hdl.handle.net/10651/52220
dc.format.extentp. 327-330
dc.language.isoeng
dc.relation.ispartofExpert Opinion on Drug Discovery, 14
dc.rights© 2019 Informa UK Limited, trading as Taylor & Francis Group
dc.rightsCC Reconocimiento - No comercial - Sin obras derivadas 4.0 Internacional
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/
dc.sourceScopus
dc.source.urihttps://www.scopus.com/inward/record.uri?eid=2-s2.0-85062969477&doi=10.1080%2f17460441.2019.1581169&partnerID=40&md5=e63fe55248a5a2396fa977bee3031438
dc.titleWhy have transgenic rodent models failed to successfully mimic Alzheimer’s disease. How can we develop effective drugs without them?
dc.typejournal article
dc.identifier.doi10.1080/17460441.2019.1581169
dc.relation.publisherversionhttp://dx.doi.org/10.1080/17460441.2019.1581169
dc.rights.accessRightsopen access
dc.type.hasVersionAM


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