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Drug-induced hyperploidy stimulates an antitumor NK cell response mediated by NKG2D and DNAM-1 receptors

dc.contributor.authorAcebes Huerta, Andrea 
dc.contributor.authorLorenzo Herrero, Seila 
dc.contributor.authorRodríguez Folgueras, Alicia 
dc.contributor.authorHuergo Zapico, Leticia 
dc.contributor.authorLópez Larrea, Carlos 
dc.contributor.authorLópez Soto, Alejandro 
dc.contributor.authorGonzález Rodríguez, Segundo 
dc.date.accessioned2016-06-21T11:37:18Z
dc.date.available2016-06-21T11:37:18Z
dc.date.issued2016
dc.identifier.citationOncoImmunology, 5(2), p. 1-15 (2016); doi:10.1080/2162402X.2015.1074378
dc.identifier.issn2162-4011
dc.identifier.urihttp://hdl.handle.net/10651/37900
dc.description.sponsorshipThis work was supported by the Spanish grant from PI12/01280 from the Instituto de Salud Carlos III and the FEDER program of the European Community, and, in part, by the Spanish grant from the “Fondo de Investigaciones Sanitaria”, FEDER European Union (PI12/02587), and from “Red de Investigación Renal” REDinREN RD12/0021/0021.
dc.format.extentp. 1-15
dc.language.isoeng
dc.relation.ispartofOncoImmunology
dc.rights© 2016 Taylor & Francis Group, LLC
dc.sourceScopus
dc.source.urihttps://www.scopus.com/inward/record.uri?eid=2-s2.0-84959377607&partnerID=40&md5=bcbc79d2c56c1611da71e34914efb58d
dc.titleDrug-induced hyperploidy stimulates an antitumor NK cell response mediated by NKG2D and DNAM-1 receptors
dc.typejournal article
dc.identifier.doi10.1080/2162402X.2015.1074378
dc.relation.projectIDPI12/01280
dc.relation.projectIDFEDER/PI12/02587
dc.relation.projectIDRD12/0021/0021
dc.relation.publisherversionhttp://dx.doi.org/10.1080/2162402X.2015.1074378


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