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Pro-autophagic polyphenols reduce the acetylation of cytoplasmic proteins

dc.contributor.authorPietrocola, Federico
dc.contributor.authorMariño García, Guillermo 
dc.contributor.authorLissa, Delphine
dc.contributor.authorVacchelli, Erika
dc.contributor.authorMalik, Shoaib Ahmad
dc.contributor.authorNiso-Santano, Mireia
dc.contributor.authorZamzami, Naoufal
dc.contributor.authorGalluzzi, Lorenzo
dc.contributor.authorMaiuri, Maria Chiara
dc.contributor.authorKroemer, Guido
dc.date.accessioned2017-04-04T07:57:21Z
dc.date.available2017-04-04T07:57:21Z
dc.date.issued2012
dc.identifier.citationCell Cycle, 11, 20, p. 3851-3860 (2012); doi:10.4161/cc.22027
dc.identifier.issn1538-4101
dc.identifier.urihttp://hdl.handle.net/10651/41840
dc.description.sponsorshipG.K. is supported by the Ligue Nationale contre le Cancer (LNC, Equipe labelisée), Agence Nationale pour la Recherche (ANR), European Commission (Active p53, Apo-Sys, ChemoRes, ApopTrain), Fondation pour la Recherche Médicale (FRM), Institut National du Cancer (INCa), Cancéropôle Ile-de-France, Fondation Bettencourt-Schueller and the LabEx ImmunoOncology. G.M. and M.N.S. are supported by an EMBO fellowship and a postdoctoral contract from the Junta de Extremadura, respectively. S.A.K. is the recipient of a grant from the Higher Education Commission (HEC) of Pakistan. L.G. is financed by the LabEx Immuno-Oncology
dc.format.extentp. 3851-3860
dc.language.isoeng
dc.publisherTaylor and Francis Inc.
dc.relation.ispartofCell Cycle
dc.rights©
dc.sourceScopus
dc.source.urihttps://www.scopus.com/inward/record.uri?eid=2-s2.0-84868026885&doi=10.4161%2fcc.22027&partnerID=40&md5=1048ab0a356769d02c300585bd5e6da9
dc.titlePro-autophagic polyphenols reduce the acetylation of cytoplasmic proteins
dc.typejournal article
dc.identifier.doi10.4161/cc.22027
dc.relation.publisherversionhttp://dx.doi.org/10.4161/cc.22027


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